Clinical Subject Page
Abdominal aortic aneurysm (AAA)
ICD-10
I71.4
Specialty
Cardiology
Onset
Chronic
Reviewed
June 2026
On This Page
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OverviewOverview
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Etiology & Risk FactorsEtiology & Risk Factors
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PathophysiologyPathophysiology
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Clinical PresentationClinical Presentation
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History TakingHistory Taking
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Physical ExaminationPhysical Examination
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InvestigationsInvestigations
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DiagnosisDiagnosis
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ManagementManagement
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ComplicationsComplications
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PrognosisPrognosis
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Key Points / Clinical PearlsKey Points / Clinical Pearls
Overview
Abdominal aortic aneurysm (AAA) is a localized dilation of the abdominal aorta measuring ≥3.0 cm in diameter or ≥50% greater than the normal vessel diameter, resulting from weakening of the aortic wall and increasing the risk of rupture.
Teaching point
Most AAAs are asymptomatic and are detected incidentally; aneurysm diameter is the strongest predictor of rupture.
Etiology & Risk Factors
Etiology & Risk Factors
Etiology
- Atherosclerosis (most common)
- Chronic hypertension
- Smoking-related vascular degeneration
- Connective tissue disorders (Marfan, Ehlers-Danlos)
- Inflammatory aortitis
- Infectious (mycotic) aneurysm
- Previous aortic trauma
Risk Factors
- Age ≥65 years
- Male sex
- Smoking (strongest modifiable risk factor)
- Hypertension
- Family history of AAA
- Hyperlipidemia
- Coronary artery disease
- Peripheral arterial disease
Pathophysiology
- Inflammation and proteolytic degeneration of collagen, elastin, and smooth muscle cells weaken the abdominal aortic wall.
- Loss of wall strength leads to progressive widening (aneurysm).
- Mechanical stress (especially from hypertension) further enlarges the aneurysm and may cause rupture.
- The dilated vessel disrupts laminar blood flow, producing turbulent flow.
- Mural thrombi may develop within the aneurysm and cause peripheral thromboembolism.
Simple Flow
Wall degeneration → Weak abdominal aortic wall → Aneurysm formation → Turbulent blood flow → Thrombus formation ± Rupture
Clinical Presentation
Symptoms
- Usually asymptomatic
- Chest pain
- Back pain
- Hoarseness
- Dysphagia
- Dyspnea
- Cough
Signs
- Early diastolic murmur (aortic regurgitation)
- Features of connective tissue disorders
- Signs of compression of adjacent structures
- Hypotension or shock (rupture)
Important Note
The classic triad of ruptured AAA is abdominal/back pain, hypotension, and a pulsatile abdominal mass, although all three are not always present.
History Taking
Key Questions
- Sudden abdominal or back pain?
- Pulsatile abdominal sensation?
- History of smoking?
- Hypertension?
- Family history of AAA?
- Previous aneurysm?
- Syncope or dizziness?
- Peripheral vascular disease?
Red Flags
- Sudden severe abdominal or back pain
- Hypotension
- Syncope
- Pulsatile abdominal mass
- Signs of hemorrhagic shock
Physical Examination
Inspection
- May show a visible pulsation in thin patients
Palpation
- Pulsatile abdominal mass (classic finding)
Auscultation
- Abdominal bruit
General Examination
- Assess peripheral pulses
- Check for signs of hypovolemic shock if rupture is suspected
If Rupture Occurs
- Hypotension
- Tachycardia
- Severe abdominal or back pain
- Signs of shock
Investigations
Laboratory
- CBC
- Renal function
- Coagulation profile
- Blood type & crossmatch (if rupture suspected)
Imaging
- Abdominal ultrasound (screening & surveillance)
- CT angiography (gold standard for diagnosis and operative planning)
Additional Tests
- ECG (exclude cardiac causes)
- Chest X-ray (if indicated)
Diagnosis
Abdominal Aortic Aneurysm (AAA) · Diagnostic Criteria
DEFINED AS INFRARENAL AORTIC DIAMETER ≥3.0 cm (OR ≥1.5x EXPECTED NORMAL)
Most AAAs are infrarenal and asymptomatic until rupture. Diagnosis and surveillance rely on abdominal ultrasound (screening/monitoring) and CT angiography (pre-operative planning, suspected rupture). Measurements taken outer-wall-to-outer-wall, perpendicular to the aortic long axis.
Diagnostic Approach
1
Screen
One-off abdominal ultrasound screening recommended for men aged 65–75 (esp. ever-smokers). Often otherwise found incidentally on imaging or exam (pulsatile abdominal mass).
Screening
2
Confirm & Measure
Abdominal ultrasound is first-line for diagnosis/surveillance (cheap, no radiation). CT angiography for pre-operative planning or suspected rupture/symptomatic AAA.
Imaging
3
Classify & Localise
Determine extent — infrarenal (most common, ~90%), juxtarenal, suprarenal, or thoracoabdominal. Assess relationship to renal and iliac arteries for repair planning.
Anatomic mapping
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Risk Stratify
Assess size, growth rate, symptoms, and comorbidities to determine surveillance interval vs urgent surgical referral.
Integration
| Category | Diameter | Action |
|---|---|---|
| Normal infrarenal aorta | <3.0 cm | No follow-up required |
| Small AAA | 3.0–4.4 cm | Annual ultrasound surveillance; address cardiovascular risk factors |
| Medium AAA | 4.5–5.4 cm | Ultrasound surveillance every 3 months; vascular surgery referral |
| Large AAA | ≥5.5 cm | Elective surgical repair indicated (open or EVAR) regardless of symptoms |
| Rapid expansion | ≥1 cm/year or ≥0.5 cm in 6 months | Treat as high risk — expedite surgical referral regardless of absolute size |
| Symptomatic AAA (pain, tenderness) | Any size | Urgent CT angiography and surgical referral — treat as impending rupture |
| Ruptured AAA | Any size | Surgical emergency — classic triad: abdominal/back pain, pulsatile mass, hypotension |
Risk Factors
Smoking — strongest modifiable risk factor
Age >65 and male sex — incidence rises sharply with age, ~4–6x more common in men
Hypertension & atherosclerosis — chronic wall stress and degenerative change
Family history — first-degree relative with AAA increases risk significantly
Connective tissue disease — Marfan, Ehlers-Danlos (less common cause than for TAA)
COPD & peripheral arterial disease — shared atherosclerotic risk profile
Screening & Surveillance Notes
One-off USS screening — recommended for men at age 65 in many national programmes (e.g. NHS AAA screening)
Average growth rate — ~0.2–0.3 cm/year, faster as diameter increases
Repair decision — based on diameter threshold, growth rate, and patient fitness for surgery, not symptoms alone
EVAR vs open repair — EVAR has lower perioperative mortality but requires suitable anatomy and lifelong surveillance for endoleak
Smoking cessation — single most effective intervention to slow growth and reduce rupture risk
Home / Home / Clinical Cases / Cardiovascular System Clinical Cases / Abdominal Aortic Aneurysm (AAA)
Related Topics
Plaque
Management
Medical Management
- Smoking cessation
- Blood pressure control
- Statin therapy
- Antiplatelet therapy (if indicated)
- Regular imaging surveillance for small aneurysms
Surgical Management
- Endovascular aneurysm repair (EVAR)
- Open surgical repair
- Immediate repair for ruptured AAA
- Elective repair for symptomatic or large aneurysms (typically ≥5.5 cm in men, or rapidly expanding aneurysms)
Complications
- Aneurysm rupture
- Massive hemorrhage
- Thromboembolism
- Distal limb ischemia
- Aortic dissection (rare)
- Death
Prognosis
- Small AAAs have a good prognosis with surveillance.
- Risk of rupture increases markedly with aneurysm size and expansion rate.
- Ruptured AAA has a very high mortality despite emergency surgery.
Key Points / Clinical Pearls
- Smoking is the strongest modifiable risk factor for AAA.
- Ultrasound is the preferred screening test.
- CT angiography is the imaging modality of choice before repair.
- AAA is defined as an abdominal aortic diameter ≥3.0 cm.
- Elective repair is generally recommended at ≥5.5 cm in men or for symptomatic/rapidly expanding aneurysms.
- Ruptured AAA is a surgical emergency with high mortality.