Pleural Effusion
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1- Definition & Types
2- Causes (Aetiology)
3- Pathophysiology
4- Clinical Features & Examination
5- Investigations
6- Management
7- Complications
8- Core Concepts
Definition & Types
Pleural effusion is the accumulation of excess fluid in the pleural space between the visceral and parietal pleura.
Types of Pleural Effusion (based on pleural fluid analysis):
Transudate: Low protein and LDH. Caused by systemic factors such as increased hydrostatic pressure or decreased oncotic pressure.
Exudate: High protein and LDH. Caused by local factors such as inflammation, infection, or malignancy.
Light’s Criteria (used to identify exudates):
Pleural fluid is an exudate if one or more of the following are true:
Pleural fluid protein/serum protein ratio > 0.5
Pleural fluid LDH/serum LDH ratio > 0.6
Pleural fluid LDH > two-thirds of the upper limit of normal serum LDH

Causes (Aetiology)
Transudative Effusions:
Congestive heart failure
Cirrhosis (hepatic hydrothorax)
Nephrotic syndrome
Hypoalbuminaemia
Peritoneal dialysis
Exudative Effusions:
Parapneumonic effusion (due to pneumonia)
Tuberculosis
Malignancy (especially lung and breast cancer, lymphoma)
Pulmonary embolism
Connective tissue diseases (e.g. rheumatoid arthritis, systemic lupus erythematosus)
Trauma
Post-surgical or post-radiotherapy
Pathophysiology
Pleural fluid homeostasis is maintained by a balance between production and absorption. This balance can be disrupted by:
Increased hydrostatic pressure (e.g. in heart failure)
Decreased oncotic pressure (e.g. in nephrotic syndrome, hypoalbuminaemia)
Increased capillary permeability (e.g. in inflammation, infection)
Impaired lymphatic drainage (e.g. in malignancy)
The resulting accumulation of fluid impairs lung expansion and gas exchange.
Clinical Features & Examination
Symptoms:
Progressive breathlessness
Pleuritic chest pain
Non-productive cough
Often asymptomatic if the effusion is small
Signs:
Decreased chest expansion on the affected side
Stony dullness to percussion over the fluid
Diminished or absent breath sounds
Decreased tactile vocal fremitus
Tracheal deviation away from the affected side (in large effusions)
Reduced vocal resonance

Investigations
Imaging:
Chest X-ray: Blunting of the costophrenic angle, fluid level, mediastinal shift if large
Ultrasound: Sensitive for small effusions; guides safe thoracocentesis
CT thorax: Useful if malignancy or complex pathology is suspected
Pleural Fluid Analysis (after thoracocentesis):
Appearance (clear, turbid, purulent, bloody)
Protein, LDH, glucose, pH
Cytology for malignant cells
Gram stain, culture, and acid-fast bacilli testing (if TB suspected)
Blood tests:
Serum protein and LDH (for Light’s criteria comparison)
Full blood count, CRP/ESR
Renal and liver function tests
Autoantibodies if connective tissue disease suspected
Management
Underlying Cause:
Heart failure: diuretics
Infection: appropriate antibiotics
Malignancy: oncological therapy ± pleurodesis
Symptomatic Relief:
Therapeutic thoracocentesis: improves breathlessness
Chest drain: if effusion is large, recurrent, or purulent (empyema)
Pleurodesis: for recurrent malignant effusion (usually talc)
Surgical options:
Video-assisted thoracoscopic surgery (VATS) for biopsy or decortication in empyema
Complications
Respiratory compromise
Infection leading to empyema
Pneumothorax (iatrogenic)
Fibrosis or trapped lung
Reaccumulation of fluid
Septation or loculated effusion
Core Concepts
Light’s criteria are essential for distinguishing transudates from exudates.
Bilateral effusions are more likely to be transudative; unilateral effusions should prompt diagnostic thoracocentesis.
Tuberculosis and malignancy are common causes of unilateral exudative effusions.
Always consider ultrasound guidance before pleural procedures to minimise complications.
Low pleural fluid glucose or pH suggests infection, malignancy, or rheumatoid effusion.