DiGeorge Syndrome
Content of This Page
1- Overview and Genetic Basis
2-Embryological Defect
3- Clinical Features
4- Congenital Heart Defects
5– Diagnosis and Investigations
6- Management Principles
Overview and Genetic Basis
Definition: A congenital syndrome caused by a microdeletion on chromosome 22q11.2.
Inheritance: Most cases are sporadic, but it can be autosomal dominant.
Syndromic features arise due to failure of development of the 3rd and 4th pharyngeal pouches.
Also known as: 22q11.2 deletion syndrome or velocardiofacial syndrome (variant presentation).

Embryological Defect
Affects derivatives of the 3rd pharyngeal pouch (thymus, inferior parathyroids)
and 4th pharyngeal pouch (superior parathyroids).Leads to:
Hypoplastic or absent thymus → impaired T-cell immunity
Absent or underdeveloped parathyroid glands → hypocalcaemia
Craniofacial and cardiac anomalies
-Mnemonic: “CATCH-22”
Cardiac defects, Abnormal facies, Thymic hypoplasia, Cleft palate, Hypocalcaemia (22q11 deletion)
Clinical Features
Immunodeficiency: Due to T-cell dysfunction (recurrent viral/fungal infections)
Hypocalcaemia: Neonatal tetany, seizures (from low PTH)
Congenital heart defects: Especially conotruncal malformations
Facial dysmorphism:
Low-set ears
Micrognathia (small jaw)
Short philtrum
Hypertelorism (wide-set eyes)
Cleft palate or velopharyngeal insufficiency
Developmental delay, learning difficulties, and later psychiatric disorders (e.g. schizophrenia risk)
Congenital Heart Defects
Occur in ~75–80% of patients.
Typically involve outflow tract anomalies:
Tetralogy of Fallot
Interrupted aortic arch
Truncus arteriosus
Ventricular septal defect (VSD)
Heart defects may be the first clue prompting genetic testing.
Diagnosis and Investigations
Genetic testing:
FISH (fluorescence in situ hybridisation) or
Chromosomal microarray confirms 22q11.2 deletion
Blood tests:
↓ Total calcium and ↓ parathyroid hormone (PTH)
↓ Absolute T-cell count
Chest X-ray:
May show absent thymic shadow
Immunological assessment:
Evaluate T-cell function
Ig levels may be normal or low (secondary B-cell dysfunction)

Management Principles
Hypocalcaemia:
Treat with calcium and vitamin D
Immunodeficiency:
Avoid live vaccines (if T-cell deficient)
Give prophylactic antibiotics or immunoglobulin if indicated
Cardiac surgery: Correct congenital heart defects early
Speech and developmental therapy
Genetic counselling for families
Lifelong follow-up due to risk of autoimmune disease, psychiatric illness, and endocrine problems